You spoke to a potential mechanism of action of the injury.
And you mentioned to me the use of a D-dimer test, which we all know what it's for, but we're going to have to explain a little bit.
Could you please speak to this a little bit and give the relevant context and introduction, please?
Because I think this is really groundbreaking and important.
Yes, yeah, thank you. So, so, so one of the the key things
that really bothered me when I started to see vaccine, serious
vaccine injuries in my own patients is that I had no idea
what the mechanism of injury was and therefore, as their
doctor, I had no idea how to treat it. Because, you know, as
their family doctor, um
I, I need, they would come to me for help and I needed to help them and I, and I was clueless.
I mean this was, this is an experiment and I was aware that there was literally,
what we call iatrogenic disease, a medically induced
disease being produced by this vaccine. And so I had asked this in my open letter to Dr. Bonnie Henry,
our provincial health officer, what is the mechanism of injury and how do I treat this
as these people's doctor? And of course, nobody knew. And the vaccine manufacturers had told us
that the COVID spike protein does not go intravenous. It stays in the arm.
The antibodies to the spike protein are produced in the arm, and that's what we have been found.
But scientists now, and Dr Brody has actually very clearly revealed this, that only 25 percent
of the vaccine actually stays in the arm. And the rest of it, so these vaccines are a
a vast number of little messenger RNA strands. The Moderna vaccine has 40 trillion messenger RNA
molecules per vaccine dose. 40 trillion. So.
So, these are wrapped in a little lipid capsule. The lipid capsule is to enable them to be absorbed into the cells. So this is injected into the person's arm and their deltoid muscle of the shoulder.
From there, as I mentioned, only 25% actually stays there. The rest is taken up collected through the lymphatic system and fed into the general circulation.
around the entire body and I think every doctor knows that absorption from the the circulation
occurs in capillary networks because that's where the blood slows right down. It's going through
tiny tiny vessels so these little nano capsules containing these trillions of messenger RNA
molecules are absorbed in
the lining around the capillaries, what medically we call the vascular endothelium. So these little
cup these little packages are
absorbed into the cells around the vessels, the packages open, the body recognizes these
messenger RNA strands as a gene, and gets to work making COVID spike proteins. So in
a virus, those COVID spike proteins form part of the viral capsule. But the problem is they're
not in a virus, they're in
they're in the cells around blood vessels. So as a result, they become part of the cell wall
of that cell. So normally, the cells that surround your blood vessels have to be very, very smooth
to enable good and unimpeded flow of blood. But as soon as you've got all these little spike
proteins that become part of the cell wall,
It's now a rough surface. It's going to be like a very coarse sandpaper.
It's now what the platelets are going to interpret as a damaged vessel. It's no longer smooth.
rough. So clotting is inevitable because the platelets that come down that vessel are going
to hit a rough spot and assume this must be a damaged vessel. This vessel needs to be blocked
to stop the bleeding. That's how our clotting works. So, so, so clots are according because
of this and because of the nature of this clots are in
because of these spike proteins in the capillary networks.
So I set out to then try and prove this.
it could this theory be correct.
And so the problem is these little clots
in the capillary networks are microscopic
and they are scattered.
So they're not going to show on any scan.
They're just too small and too scattered.
It's not like the big clots that cause strokes
or heart attacks.
They're too small and they're too scattered.
So how on earth can we know if the person clotted?
And the only way is with a blood test called
D-dimer. So the D-dimer is a blood test that will show up a recent clot. It won't show up an old clot, it shows up a new clot.
And it doesn't tell you where the clot is, it just tells you that the clotting mechanism has been activated.
So I have now been recruiting patients from my practice, people that have come into my office and others that have heard me speak about this and have asked people to do this D-dimer within one week of their COVID shot.
And the study is ongoing. These are preliminary results. So far I've got 62% positive elevated D Dimer, which means that the blood clots are not rare.
That's what the so-called experts keep telling us. The clots are rare.
are rare, but the small ones are clearly happening in the majority of people, 62 percent. Now I'll
tell you what the real concern with this is, is that a clotted vessel is permanently damaged.
That vessel never ever goes back to normal.
So if this theory is correct, which it really looks like by these D-dimer results, and I'm told it has been done in Australia, and it's been done in the UK, and they also found elevated D-dimers.
And they sort of discarded the information because they said there's no clinical evidence of plots.
Well, the clinical is meaningless because they're microscopic and they're scattered, and so you're not going to see clinical evidence.
But in fact, all of the...
The frequent side effects of the shot, which are headache, nausea, dizziness, fatigue, could all be signs of cerebral thrombosis on a capillary level.
I mean, those literally, you could be having...
thousands and thousands of tiny tiny little clots in your brain that weren't showing a scan, but they will give you those exact symptoms.
So, so the concern is I have now got six people in my medical practice that that cannot exert themselves the way they used to be what what medically we call reduced effort tolerance.
Six people who who now get out of breath doing things that they could previously do without any problem.
So I believe that these people locked up.
thousands and thousands of capillaries in their lungs,
in these six people.
So I believe these people now have permanently damaged lungs
because they have got,
I mean, and that's why they get out of breath.
I have one fellow that used to walk two miles
to my office every week for a shock for his arthritis.
And he says after a quarter of a mile, he's done.
In other words, his effort tolerance is reduced
to one eighth of what it used to be.
And so I've sent some of these people for chest x-rays and CT scans to see what it shows.
And all it shows is distorted architecture.
What the Radiolus record describes increased reticulation.
It's a very nonspecific thing.
And it's because it's microscopic.
But the concern is, because these vessels are now
permanently damaged in a person's lungs.
When the heart tries to pump blood
through all those damaged vessels,
there's increased resistance
trying to pump the blood through those lungs.
So those people are going to develop something
called Pomeroy artery hypertension,
high blood pressure in their lungs.
And the concern with that
is that those people will probably all develop right-sided heart failure within three years and
die because they now have increased vascular resistance through their lungs and lung tissue
heart tissue and brain and spinal tissue,
all of that does not regenerate.
In other tissues it can regenerate liver and kidneys
and muscle and other,
but there's some tissues that cannot.
And so this absolutely explains what I've seen
in my patients and that's what I'm doing
to prove it in my studies ongoing.